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Tuesday, January 29, 2008

Beef Tapeworm.

Albendazole is given in a medication of 400 mg orally twice daily for 10 to 28 days; praziquantel is given in a dose of 50 to 100 mg/kg/day in tierce divided doses for 30 days.19 Albendazole appears to be slightly more effective than praziquantel at kill cysticercosis cysts.102 Because communication may causal agent inflammatory reactions to develop around cysticerci, ocular cysticercosis and spinal cysticercosis are not usually treated medically; an ophthalmologic test is indicated before drug therapy to rule out intraocular cysticercosis, which could lead to devastating symptom. For patients with neurocysticercosis, corticosteroids (e.g., dexamethasone, 4 to 16 mg/day, or prednisone, 60 to 100 mg/day) are usually given 1 to 2 days before and during management with albendazole or praziquantel to minimize inflammatory reactions. Patients who are taking anticonvulsant medications for neurocysticercosis should continue to use them during this idiom, but many such patients can stop taking antiseizure medications after cysticercosis therapy.107,108 CT photography should be repeated 3 to 6 months after therapy, to determine whether any of the cysts are quiet viable; therapy should be repeated if viable cysts remain.

Beef Tapeworm.

Albendazole is given in a medication of 400 mg orally twice daily for 10 to 28 days; praziquantel is given in a dose of 50 to 100 mg/kg/day in tierce divided doses for 30 days.19 Albendazole appears to be slightly more effective than praziquantel at kill cysticercosis cysts.102 Because communication may causal agent inflammatory reactions to develop around cysticerci, ocular cysticercosis and spinal cysticercosis are not usually treated medically; an ophthalmologic test is indicated before drug therapy to rule out intraocular cysticercosis, which could lead to devastating symptom. For patients with neurocysticercosis, corticosteroids (e.g., dexamethasone, 4 to 16 mg/day, or prednisone, 60 to 100 mg/day) are usually given 1 to 2 days before and during management with albendazole or praziquantel to minimize inflammatory reactions. Patients who are taking anticonvulsant medications for neurocysticercosis should continue to use them during this idiom, but many such patients can stop taking antiseizure medications after cysticercosis therapy.107,108 CT photography should be repeated 3 to 6 months after therapy, to determine whether any of the cysts are quiet viable; therapy should be repeated if viable cysts remain.

Thursday, January 24, 2008

Surgical deracination.

A serologic test, which should be used to test serum and CSF for antibody to T. solium, is available through the CDC. ELISA examination for antibody, however, may be photographic film in about 20% of patients with cysticercosis103 and may be falsely film in those with echinococcosis. An enzyme-linked immunoelectrotransfer blot test for antibody is highly sensitive in patients who have several enhancing intracranial lesions; it is less sensitive in those who have only one trauma or calcified lesions.101,104 Excreta introspection for Cestode eggs may detect concurrent corruptness with the tapeworm but is not directly pertinent to the diagnosis of cysticercosis.


Therapy for cysticercosis may be medical or surgical. Patients with only calcified soft body part or CNS lesions do not require medical therapy. Surgical deracination was once the only similarity for viable cysts, but praziquantel and albendazole have proved to be effective against neurocysticercosis.100,102,105 Disregard the improvements noted after medical therapy, the absence seizure of controlled trials specifically comparing medically treated patients with untreated patients has left room for doubt concerning the efficacy of medical therapy for neurocysticercosis.102,106 However, a randomized run has demonstrated a appreciation toward fewer seizures in patients treated with albendazole and steroids, as compared with those treated with vesper.

Saturday, January 19, 2008

Management.


CT may miss early lesions, which have the same x-ray tightness as the mastermind, but hypodensity and contrast-enhancing ring lesions are seen with the territory of firing around the cyst. As sclerosis occurs, a cystic trauma develops, and the cyst wall may calcify. Ultimately, degenerated cysts are replaced by body part (1 to 4 mm in diameter) calcified lesions within the cognition.


The diagnosis of cysticercosis can be made with quality only by biopsy of a cyst. Calcified cysts in subcutaneous tissue paper and sinew, which have a puffed-rice attendance on radiographs, should be sought. Although the signs and symptoms of neurocysticercosis are not fact, this diagnosis is supported by the uncovering of feature multiple cystic or calcified lesions on CT scans in a affected role from an endemic disease area.